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Cardiac Glycosides & Antidysrhythmic Class I-II - Pharmacology - Cardiovascular | @LevelUpRN Side Effects 0 Views • 2 months ago

Cathy covers the following cardiovascular medications in this video: Cardiac Glycosides (Digoxin), Class I Antidysrhythmics (sodium channel blockers), and Class II Antidysrhythmics (beta blockers).

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0:00 What To Expect
0:15 Cardiac Glycosides
0:31 Digoxin
0:45 Mode of Action
1:04 Side Effects
1:41 Administration
1:56 Nursing Care
3:01 Class I Antidysrhythmics
3:38 Side Effects/ Black Box Warning
4:12 Nursing Care
4:29 Class II Antidysrhythmics
4:57 Side Effects
5:28 Beta 1 vs. Non Selective Blockers
6:14 What’s Next?

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Pharmacology: Drugs for Heart Failure, Animation Side Effects 0 Views • 2 months ago

Mechanisms of actions of ACE inhibitors, Angiotensin receptor blockers (ARBs), Beta-blockers, Aldosterone receptor antagonists (ARAs), Digoxin, Ivabradine, Angiotensin Receptor-Neprilysin Inhibitor, ARNIs (sacubitril/valsartan), Diuretics, Vasodilators. Role of RAAS, sympathetic system and natriuretic peptide system in pathophysiology of Congestive heart failure (CHF) and rational for treatment of HF.

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Heart failure is when the heart is unable to pump effectively, called systolic heart failure; or unable to fill properly, called diastolic heart failure. In both cases, blood output is reduced. Ejection fraction is reduced in systolic heart failure, but typically preserved in diastolic heart failure.
The pathophysiology of heart failure involves a vicious cycle in which reduced cardiac output, as a compensatory response, activates the renin-angiotensin-aldosterone system (RAAS) and sympathetic system. However, these systems cause vasoconstriction, increase heart rate and blood pressure, making it even harder for the heart to pump. Increased aldosterone level also promotes ventricular remodeling, myocardial scarring, and vascular injury, worsening the disease.
On the other hand, the natriuretic peptide system is also activated. This system is protective to the heart. It promotes vasodilation, sodium and water excretion, and inhibits cardiac remodeling.
Most drugs used in heart failure therapy aim to inhibit RAAS and sympathetic activities, and/or promote the natriuretic system. Other drugs increase ventricular contractility or reduce water retention – a major heart failure symptom.
First-line therapy for patients with reduced ejection fraction typically includes an angiotensin-converting enzyme (ACE) inhibitor, and a beta-blocker.
- ACE inhibitors block the conversion of angiotensin-I to angiotensin-II in RAAS, thereby inhibiting RAAS activity. Common side effects include dry cough, headache, and hypotension. Rarely, ACE inhibitors may cause a swelling reaction known as angioedema.
- Angiotensin receptor blockers (ARBs) inhibit the effects of angiotensin-II. Their mechanism of action is similar to that of ACE inhibitors, but they do not usually cause a cough.
- Beta-blockers decrease heart rate by binding to β1-adrenergic receptor in the heart and blocking the sympathetic influences that act through these receptors. Adverse effects: hypotension, bradycardia and AV blocks.
- Vasodilators reduce blood pressure and are usually used for patients who cannot tolerate ACE inhibitors or ARBs.
- Diuretics are often prescribed to relieve fluid retention. Loop diuretics are most powerful and typically used for most patients. Thiazides are less effective, but they also have a vasodilation effect, and are thus preferred for patients with hypertension but only mild fluid retention.
- Aldosterone receptor antagonists block the action of aldosterone. Because aldosterone’s primary function is to promote sodium and water retention, and potassium excretion; aldosterone antagonists act as potassium-sparing diuretics. However, their effect in heart failure treatment is also attributed to the inhibition of aldosterone’s damaging impact on the heart and blood vessels.
- Digoxin increases cardiac contractility by inhibiting the sodium-potassium pump, causing intracellular sodium concentration to rise. This then leads to higher levels of intracellular calcium via the action of sodium-calcium exchanger. Higher calcium results in increased muscle contraction. Digoxin also decreases sympathetic activities, slowing down heart rate.
- Ivabradine slows the heart rate by inhibiting the “funny” channel responsible for spontaneous firing of the SA node. Adverse effects include bradycardia, atrial fibrillation, and vision problems.
- ARNIs are a new class of medications. ARNI therapy consists of a neprilysin inhibitor and an ARB.

Digoxin Toxicity - Symptoms, Diagnosis & Treatment | Credihealth Side Effects 0 Views • 2 months ago

Digoxin Toxicity - Symptoms, Diagnosis & Treatment | Credihealth

Digoxin toxicity is a potentially serious condition that can occur when there are excessive levels of the medication digoxin in the body. Digoxin is a cardiac glycoside primarily used to treat various heart conditions, including heart failure, atrial fibrillation, and other supraventricular tachycardias.

The signs and symptoms of digoxin toxicity may include:

Cardiac effects:
Bradycardia (slow heart rate)
Irregular heartbeats (arrhythmias)
Cardiac conduction disturbances
Gastrointestinal effects:
Nausea and vomiting
Abdominal pain
Diarrhea
Anorexia

Neurological effects:
Visual disturbances (blurred vision, yellow-green halos)
Confusion
Drowsiness
Headache
Dizziness
Weakness

Other effects:
Electrolyte imbalances (hypokalemia, hypercalcemia)
Increased risk of digoxin toxicity in patients with kidney dysfunction
The risk of digoxin toxicity increases with factors such as:

Excessive dosage of digoxin
Impaired kidney function (reduced clearance of digoxin)
Electrolyte imbalances (hypokalemia, hypercalcemia, hypomagnesemia)
Drug interactions (e.g., with diuretics, antiarrhythmics, antibiotics)
Advanced age
Low lean body mass

If digoxin toxicity is suspected, immediate medical attention is required. Treatment may involve discontinuing digoxin, administering digoxin-specific antibody fragments (Digibind) to bind and remove digoxin from the body, correcting electrolyte imbalances, and providing supportive care as needed.

Monitoring digoxin levels, adjusting dosages based on kidney function, and avoiding drug interactions are essential measures to prevent digoxin toxicity in patients receiving this medication.

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Digoxin: Use & Effects in Heart Failure (Part 1) | Sketchy Side Effects 0 Views • 2 months ago

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An overview of digoxin, a cardiac glycoside used for symptomatic treatment of chronic heart failure. It explains how digoxin works, the drug's role as an antiarrhythmic and its potential side effects, such as hyperkalemia and gastrointestinal symptoms.

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Digitalis | Digoxin Clinical Uses and Adverse Effects Side Effects 0 Views • 2 months ago

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Digitalis | Digoxin Clinical Uses and Adverse Effects

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Digoxin is used to treat heart failure and abnormal heart rhythms (arrhythmias). It helps the heart work better and it helps control your heart rate.
Digoxin is a medication used to manage and treat heart failure and certain arrhythmias, and abortion. It is in the cardiac glycoside class of drugs. This article outlines the indications, action, and contraindications for digoxin as a valuable agent in managing heart failure and certain arrhythmias
Digoxin is used to treat heart failure, usually along with other medications. It is also used to treat certain types of irregular heartbeat (such as chronic atrial fibrillation). Treating heart failure may help maintain your ability to walk and exercise and may improve the strength of your heart. Treating an irregular heartbeat can also improve your ability to exercise.Digoxin belongs to a class of medications called cardiac glycosides. It works by affecting certain minerals (sodium and potassium) inside heart cells. This reduces strain on the heart and helps it maintain a normal, steady, and strong heartbeat.

How to use Digoxin
Take this medication by mouth with or without food as directed by your doctor, usually once daily. If you are using the liquid form of this medication, carefully measure the dose using the dropper provided by the manufacturer. Do not use a household spoon because you may not get the correct dose.

Your body may not absorb this drug as well if you also eat foods that are high in fiber or if you take certain medications. Take this medication at least 2 hours before or after eating food products that are high in fiber (such as bran). If you are also taking cholestyramine, colestipol, or psyllium, wait at least 2 hours after taking your digoxin dose before taking any of these products. If you are taking antacids, kaolin-pectin, milk of magnesia, metoclopramide, sulfasalazine, or aminosalicylic acid, take them as far apart from your digoxin dose as possible. Ask your pharmacist if you are not sure when to take any of your medications.

The dosage of this medication is based on your medical condition, age, body weight, laboratory tests, and response to treatment.

Use this medication regularly to get the most benefit from it. To help you remember, use it at the same time each day. Do not stop taking this medication without consulting your doctor. Some conditions may become worse when the drug is suddenly stopped.
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Digoxin Mnemonic - Cardiac Glycosides (Inotropes) - Cardiac Pharmacology - Cardiology Side Effects 0 Views • 2 months ago

Digoxin (Digitalis or Cardiac glycosides include Digoxin, Digitoxin, Oubain) Mnemonic | Cardiology.

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Digoxin Explained Clearly - Exam Practice Question Side Effects 0 Views • 2 months ago

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Dr. Roger Seheult reviews key features of digoxin and how it relates to exam questions and clinical practice. Includes discussion on the digoxin mechanism of action and narrow therapeutic window, how antacids affect digoxin, and digoxin side effects and toxicity. This question is from the BoardVitals NAPLEX Question Bank.

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