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Does Azilect Cause Nausea? - Everyday Parkinsons Help Side Effects 2 Views • 1 month ago

Does Azilect Cause Nausea? In this informative video, we will discuss the medication Azilect, also known as rasagiline, which is commonly prescribed for individuals living with Parkinson’s disease. Understanding the potential side effects of any medication is important for managing health effectively. This video will focus specifically on nausea, a side effect that some patients may experience while taking Azilect. We will cover the nature of this side effect, how it can vary from person to person, and what steps should be taken if nausea becomes severe or persistent.

Additionally, we will touch on other gastrointestinal symptoms that may accompany Azilect use and the importance of monitoring these effects. Open communication with healthcare providers is essential for anyone using this medication, as it allows for adjustments and ensures the treatment remains effective.

Whether you are a patient, caregiver, or simply seeking information about Parkinson’s disease treatments, this video aims to provide clarity and guidance. Join us as we navigate the topic of Azilect and its effects, and make sure to subscribe for more helpful discussions on managing Parkinson’s disease and related treatments.

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#Azilect #Rasagiline #ParkinsonsDisease #Nausea #MedicationSideEffects #Healthcare #PatientCare #GastrointestinalHealth #ParkinsonsTreatment #HealthManagement #MedicationMonitoring #ParkinsonsSupport #ChronicIllness #CaregiverSupport #HealthAwareness

About Us: Welcome to Everyday Parkinsons Help! This channel is dedicated to providing support for individuals and families affected by Parkinson's Disease. Our mission is to share information on key topics such as Parkinson’s disease symptoms, early signs of Parkinson’s, Parkinson’s diagnosis, tremors and movement disorders, and living with Parkinson’s. We also discuss Parkinson’s medications, stages of the disease, physical therapy options, and advances like deep brain stimulation. The content provided is for general informational and educational purposes only. It is not intended to substitute for professional medical advice, diagnosis, or treatment. Never disregard professional medical advice or delay seeking it because of something you have seen in this content. Never rely on this information in place of consulting with qualified healthcare professionals. The creators and distributors of this content are not responsible for any adverse effects or consequences resulting from the use of any suggestions, preparations, or procedures described in this material. Always consult with your healthcare provider before starting any new health-related practice or program.

Warfarin: Information About Warfarin | Warfarin Interactions | Warfarin Side Effects Coumadin Side Effects 2 Views • 1 month ago

Coumadin: Information About Coumadin For Patients | Coumadin Interactions | Coumadin Side Effects | Warfarin Patient Information | Warfarin Food Drug Interactions

Hey guys! This weeks video is a short guide on warfarin also known as (coumadin). I see many patients on warfarin everyday and they are very knowledgable about their medicine, which is fantastic! But here's some information about warfarin, how it works, international normalised ratio (INR) and interactions which you may find useful.

WHAT IS WARFARIN:
Warfarin is the main oral anticoagulant used in the UK. An anticoagulant is a medicine that prevents blood clotting.

Clotting (thickening) is a complex process involving a number of substances called clotting factors.

Clotting factors are produced by the liver and help control bleeding. They work with cells that trigger the clotting process (platelets) to ensure blood clots effectively.

Warfarin blocks one of the enzymes (proteins) that uses vitamin K to produce clotting factors. This disrupts the clotting process, making it take longer for the blood to clot.

WHEN IS WARFARIN PRESCRIBED:
Anticoagulant medicines, such as warfarin, are often prescribed for people who've had a condition caused by a blood clot or have an increased risk of developing harmful blood clots.

TAKING WARFARIN:
It's very important that you take warfarin exactly as directed. Don't increase your prescribed dose unless the doctor in charge of your care advises you to.

Warfarin is taken once a day, usually in the evening. It's important to take your dose at the same time each day, before, during or after a meal.

The aim of warfarin therapy is to decrease the blood's tendency to clot, but not stop it clotting completely. This means the dose of warfarin you're taking must be carefully monitored and, if necessary, adjusted.

You'll have regular blood tests at your GP surgery or local anticoagulant clinic to make sure your dose is correct.

The INR is a measure of how long it takes your blood to clot. When you start taking warfarin, you may be given a yellow booklet about anticoagulants, which explains your treatment.

INTERACTIONS WITH WARFARIN

Medicines
Warfarin can interact with many other medicines, herbal medicines and supplements. Always ask your pharmacist, GP or staff at your anticoagulant clinic before you take them as they may interact with your warfarin.

Also visit https://bnf.nice.org.uk/interaction/warfarin.html to check medication interactions.

Foods and drink
Foods containing large amounts of vitamin K include:

• Green leafy vegetables, such as broccoli and spinach
• Vegetable oils
• Cereal grains
• Small amounts of vitamin K can also be found in meat and dairy foods.

When your first dose of warfarin is prescribed, it doesn't matter how much vitamin K you're eating because the dosage will be based on your current blood clotting levels.

However, if you make significant changes to your diet, such as increasing your vitamin K intake or cutting out foods that contain vitamin K, it could interfere with how warfarin works.

Consult the healthcare professional responsible for your care before making any significant changes to your diet while taking warfarin. Why you should avoid cranberry juice whilst taking warfarin is in the link lower down.

Alcohol
Getting drunk or binge drinking is dangerous while taking warfarin. It may increase the effect of the drug, increasing the risk of bleeding.

See links below for more information.

SIDE EFFECTS , WHEN TO SEEK MEDICAL ATTENTION & MORE INFORMATION:
Visit the following links,
https://www.nhs.uk/conditions/warfarin/
https://patient.info/medicine/....warfarin-an-anticoag

ONLINE YELLOW BOOK LINK:
http://www.npsa.nhs.uk/EasySit....eWeb/GatewayLink.asp

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DISCLAIMER:
This video is for information only and should not be used for the diagnosis or treatment of medical conditions. Abraham The Pharmacist has used all reasonable care in compiling the information but make no warranty as to its accuracy. Always consult a doctor or other healthcare professional for diagnosis and treatment of medical conditions.

Skyrizi 101: Uses, Dosing, Side Effects, Cost Concerns (Plus - Does It Really Cause Weight Loss?) Side Effects 1 Views • 1 month ago

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Skyrizi is used to treat psoriasis.

It’s mechanism of action is that it's an interleukin blocker. What that means is that eventually it's going to decrease the number of cytokines and chemokines that are produced in your body.

These substances are basically signaling proteins that lead to inflammation in your body.

Dosage

Skyrizi has a unique dosing schedule.

First of all Skyrizi comes in a 75 mg strength that you inject subcutaneously. You give two injections or 150 mg on day one, then you give 150 mg 4 weeks later, then you start giving 150 mg every 12 weeks.

The first month of dosing is what's referred to as a “loading dose”. The idea is to get your blood levels up a little higher earlier on and then level you out with the every 12 week dosing

Side Effects

Skyrizi does have some side effects that you need to be aware of.

First of all you can develop antibodies to use in Skyizi. This means that your body begins to develop antibodies to the drug and it can become less effective the longer you use it. You can see this in up to 24% of people who use it.

About 14% of people who use it can develop neutralizing antibodies. These types of antibodies actually make the drug ineffective.

The other type of side effect you can see is higher infection rates.

Remember Skyrizi acts on chemokines and cytokines. While they are involved in the inflammatory process remember that inflammation isn't always a bad thing. Part of your immune response involves inflammation. So when you knock that down it can lead to more infections.

You can see higher infection rate in up to 22% of users. Some of the more common infections tend to be upper respiratory tract infections and fungal infections.

I also warn people that you can see headaches in a small group of users as well as fatigue. But again these are only 3% to 4% of users.

One of the other things I get asked is if Skyrizi can cause weight loss?

It's actually not listed as one of the potential side effects for the drug. But keep in mind if you find that you're losing weight pay attention to if you are getting any sort of infections. If you're not feeling well because of infection there can be weight loss. But as far as Skyrizi directly affecting weight loss I think it's a long shot.

Because the drug can affect your immune system your doctor is likely going to monitor your labs throughout your use. I'll also test you for tuberculosis and monitor that as well.

How Long Does It Take To Work?

Skyrizi takes 3 to 14 days to reach peak blood levels. That doesn't mean it will take that long to start working. You may notice changes sooner.

But as far as peak effectiveness you're probably going to be looking at giving yourself at least a week or two on the medication to notice a change.

One of the biggest issues you see with people who want to use Skyrizi is that it costs quite a bit of money.

Sometimes we don't have any issues getting it through insurance other times you will. If that's the case you may want to look into financial assistance.

Here's a link to Skyrizi’s financial assistance program check it out see if you qualify: https://www.abbvie.com/patient....s/patient-assistance

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DISCLAIMER:

Any information on diseases and treatments available at this channel is intended for general guidance only and must never be considered a substitute for advice provided by a doctor or other qualified healthcare professional.

Always seek the advice of your physician or other qualified health care professional with questions you may have regarding your medical condition.

Although all measures are taken to ensure that the contents of the YouTube channel is accurate and up-to-date, all information contained on it is provided ‘as is’.

I make no warranties or representations of any kind concerning the accuracy or suitability of the information contained on this channel.

Curtis Alexander may at any time and at its sole discretion change or replace the information available on this channel.

To the extent permitted by mandatory law, Curtis Alexander shall not be liable for any direct, incidental, consequential, indirect or punitive damages arising out of access to or use of any content available on this channel, including viruses, regardless of the accuracy or completeness of any such content.

I sometimes provide links to products or services that I know, like and trust. In some cases these may be affiliate links where I earn from qualifying purchases.

EZETIMIBE (ZETIA). Mechanism of action. Indications. Explained. Side Effects 1 Views • 1 month ago

00:00 Introduction
00:18 Indications
00:44 Mechanism of action
03:40 Dosage and Side effects

Ezetimibe inhibits intestinal uptake of dietary and biliary cholesterol at the level of the brush border of the intestine by interacting with the Niemann-Pick C1-like protein 1 without affecting the absorption of fat-soluble nutrients.
According to the newest ESC Guidelines for the management of dyslipidemias
Combination with ezetimibe is recommended as additional therapy to statins for pharmacological lowering of low density lipoproteins If the goals are not achieved with the maximum tolerated dose of a statin.
This position has the strongest level of recommendation.
Absorbtion of cholesterol in intestine provided by Niemann–Pick C1-like protein 1 which transport cholesterol inside the enterocyte. Further Cholesterol with triglyceride’s carry from gut in form of chylomicrons and can be up taken by liver for further processing.
Ezetimibe inhibits intestinal cholesterol absorption by selectively blocking the Niemann–Pick C1-like protein 1 in the brush border. As a result cholesterol excretes with the feces.
By inhibiting cholesterol absorption, ezetimibe reduces the amount of cholesterol delivered to the liver by chylomocrons. When the cholesterol levels in the hepatocyte decrease. It cause the activation of LDL receptor transcription on the hepatocyte. It leads to increased clearance of LDL from the blood that therefore to decreasing intraarterial cholesterol accumulation and prevent the atherosclerosis development.
Also low level of cholesterol inside of hepatocyte increase activity of HMG-CoA reductase, which stimuli cholesterol synthesis from acetyl-Coa. This is the reason why ezetimibe is used in combination with the statins which inhibit HMG-Coa reductase therefore cholesterol synthesis.

Sources which was used for preparing:

Introduction to Lipids and Lipoproteins
https://www.ncbi.nlm.nih.gov/books/NBK305896/

Balancing Cholesterol Synthesis and Absorption in the Gastrointestinal Tract
https://www.ncbi.nlm.nih.gov/p....mc/articles/PMC23908

Ezetimibe therapy: mechanism of action and clinical update
https://pubmed.ncbi.nlm.nih.gov/22910633/

2019 ESC/EAS Guidelines for the management of dyslipidaemias: lipid modification to reduce cardiovascular risk: The Task Force for the management of dyslipidaemias of the European Society of Cardiology (ESC) and European Atherosclerosis Society (EAS)
https://academic.oup.com/eurhe....artj/article/41/1/11

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Understanding TD Side Effects 2 Views • 1 month ago

See Important Safety Information below; Prescribing Information including Boxed Warning: https://bit.ly/3YhB0s9. AUSTEDO XR®/AUSTEDO® can cause depression, suicidal thoughts, or actions in patients with HD. Don’t use if you have liver problems; are taking reserpine, tetrabenazine, or valbenazine.
INDICATIONS & USAGE
AUSTEDO XR is indicated in adults for the treatment of chorea associated with Huntington's disease (HD) & for the treatment of tardive dyskinesia (TD).
IMPORTANT SAFETY INFORMATION
Depression & Suicidality in HD Patients: AUSTEDO XR can increase the risk of depression & suicidal thoughts & behavior (suicidality) in HD Patients. Balance the risks of depression & suicidality with the clinical need for treatment of chorea. Closely monitor patients for the emergence or worsening of depression, suicidality, or unusual changes in behavior. Inform patients, their caregivers, & families of the risk of depression & suicidality & instruct them to report behaviors of concern promptly to the treating physician. Exercise caution when treating patients with a history of depression or prior suicide attempts or ideation. AUSTEDO XR is contraindicated in patients who are suicidal, & in patients with untreated or inadequately treated depression.
Contraindications: AUSTEDO XR is contraindicated in HD patients who are suicidal, or have untreated or inadequately treated depression. AUSTEDO XR is also contraindicated in: patients with hepatic impairment; patients taking reserpine or within 20 days of discontinuing reserpine; patients taking monoamine oxidase inhibitors (MAOIs), or within 14 days of discontinuing MAOI therapy; & patients taking tetrabenazine or valbenazine.
Clinical Worsening & Adverse Events in HD Patients: AUSTEDO XR may cause a worsening in mood, cognition, rigidity, & functional capacity. Prescribers should periodically re-evaluate the need for AUSTEDO XR in their patients by assessing the effect on chorea & possible adverse effects.
QTc Prolongation: AUSTEDO XR may prolong the QT interval, but the degree of QT prolongation is not clinically significant when AUSTEDO XR is administered within the recommended dosage range. AUSTEDO XR should be avoided in patients with congenital long QT syndrome & in patients with a history of cardiac arrhythmias.
Neuroleptic Malignant Syndrome (NMS), a potentially fatal symptom complex reported in association with drugs that reduce dopaminergic transmission, has been observed in patients receiving tetrabenazine. The risk may be increased by concomitant use of dopamine antagonists or antipsychotics. The management of NMS should include immediate discontinuation of AUSTEDO XR; intensive symptomatic treatment & medical monitoring; & treatment of any concomitant serious medical problems.
Akathisia, Agitation, & Restlessness: AUSTEDO XR may increase the risk of akathisia, agitation, & restlessness. The risk of akathisia may be increased by concomitant use of dopamine antagonists or antipsychotics. If a patient develops akathisia, the AUSTEDO XR dose should be reduced; some patients may require discontinuation of therapy.
Parkinsonism: AUSTEDO XR may cause parkinsonism in TD or HD patients. The risk of parkinsonism may be increased by concomitant use of dopamine antagonists or antipsychotics. If a patient develops parkinsonism, the AUSTEDO XR dose should be reduced; some patients may require discontinuation of therapy.
Sedation & Somnolence: Sedation is a common dose-limiting adverse reaction of AUSTEDO XR. Patients should not perform activities requiring mental alertness, such as operating a motor vehicle or hazardous machinery, until they are on a maintenance dose of AUSTEDO XR & know how the drug affects them. Concomitant use of alcohol or other sedating drugs may have additive effects & worsen sedation & somnolence.
Hyperprolactinemia: Tetrabenazine elevates serum prolactin concentrations in humans. If there is a clinical suspicion of symptomatic hyperprolactinemia, appropriate laboratory testing should be done & consideration should be given to discontinuation of AUSTEDO XR.
Binding to Melanin-Containing Tissues: Deutetrabenazine or its metabolites bind to melanin-containing tissues & could accumulate in these tissues over time. Prescribers should be aware of the possibility of long-term ophthalmologic effects.
Common Adverse Reactions: The most common adverse reactions for AUSTEDO (greater than 8% & greater than placebo) in a controlled clinical study in HD patients were somnolence, diarrhea, dry mouth, & fatigue. The most common adverse reactions for AUSTEDO (4% & greater than placebo) in controlled clinical studies in TD patients were nasopharyngitis & insomnia. Adverse reactions with AUSTEDO XR are expected to be similar to AUSTEDO.